Occlusal trauma signs professional#

In teeth, no cavities, restoration, fracture, micro fracture or periodontitis was found. On palpation of cervical nodes on the right side, felt swollen compared to the opposite side. Aching and scared for his condition, he decided to visit an endodontist.Ĭlinical inspection was done in the area concerned: inflammation observed lower right side of the face. However, no pain relief and the inflammation of the face persist. For 5 days and metronidazole 500 mg, one tablet every 8 hours for 5 days. Three days using the drugs, he referred not to have any relief and feel worse than before, so the doctor prescribed Dicloxacillin sodium 500 mg, one tablet every 8 hrs. They decided to begin with drugs: Diclofenac sodium 100 mg, one tablet every 8 hrs Paracetamol 500 mg, one tablet every 6 hrs Procaine benzylpenicillin 600,000 IU, one every 24 hrs for 5 days, intramuscular route. He was referred to the physicians for testing cabinet with the following results: Hematocrit 49.10, hemoglobin 16.60, platelets 307.00, WBC 7.60, VDRL, Brucella, Chagas, Hepatitis B and C and HIV, all negative. Through the anamnesis he referred, to consult the dentist not finding tooth with decay or periodontal disease. REPORTSĪ 22-year-old male was presented with severe pain intensity, facial edematous in appearance in the lower right portion. The purulent exudate initially located below the periosteum, being able to pierce, producing diffuse inflammation, which if not treated in time, can form a sub mucosal or subcutaneous abscess. Symptoms and signs contain swelling, spontaneous and intense throbbing pain character, pain to percussion and palpation. When an existing chronic apical periodontitis is secondarily infected by bacteria produces what is called a “phoenix abscess”. The paired t-test showed statistically significant differences for both the human dental pulp and periodontal ligament (P = 0.02 and P < 0.001, respectively) of the teeth under occlusal trauma when compared with the values of the control group, concluding that expression of SP is increased in human periodontal ligament and dental pulp. The results with the occlusal trauma teeth experimentally induced were 45 % and 120 % greater expression of SP in the dental pulp and periodontal ligament respectively. Nociceptive fibers are particularly common in the trigeminal nerve, which innervates the periapical tissues and dental pulp, where it has detected the presence of substance P. Substance P is a decapeptide of Arg-Pro-Lys-Pro-Gln-Gln-Fen-Fen-Gly-Leu-Met-NH2 structure, especially involved in pain perception. All samples were processed and the expression of Substance P (SP) was measured by radioimmunoassay. The remaining healthy premolars were extracted without occlusal trauma and served as controls. Before extraction, was performed an experimentally induced occlusal trauma with occlusal interference by placing a block of resin on the occlusal surface, asking the patient to chew gum for 30 minutes. Ī study of 28 samples of pulp and the periodontal ligament of healthy premolars which the extraction was indicated for orthodontic reasons was done. Occlusal alterations as bruxism, malocclusion and occlusal overload can trigger periapical damage. Marcos Lopez mentions that periapical pathologies can be caused by dental trauma affecting both the coronal portion as the tooth root. The condition or clinical entity known as occlusal trauma is synonymous with occlusion trauma, traumatic occlusion, traumatogenic occlusion, periodontal traumatism, occlusal overload, among others.

These cases eliminating the occlusal trauma and performing root canal treatment have a good prognosis.

Occlusal trauma signs professional#

Based on the above stated, is presented two cases of young healthy male of 13 and 22 years who were diagnosed by the endodontist professional with periapical pathology, which was possible caused by occlusal trauma. Subsequently, the products of the necrotic pulp could pass the apical region, as well as the inflammatory and immune dynamic process producing the apical periodontitis resulting in the release of cytokines such as Interleukin 17 that is causing the activation of osteoclasts which produce bone resortion and enlargement and spreading of periapical lesions. When it is not removed the etiological factor of long duration (occlusal trauma), aseptic pulp necrosis begins by destruction of the microvascular cells, lymphatic and nerve fiber systems. The mechanical irritants such as occlusal trauma, direct release chemical mediators in the connective tissue of the dental pulp to initiate the process of inflammation as a defense mechanism that occurs in response to mechanical aggression.

The basic pathogenic process that develops due to various causes which produce pulp or periapical pathology is the inflammatory response.